Decoy receptor 3 ameliorates experimental autoimmune encephalomyelitis by directly counteracting local inflammation and downregulating Th17 cells-Reference-Cited by-同舟云学术

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Decoy receptor 3 ameliorates experimental autoimmune encephalomyelitis by directly counteracting local inflammation and downregulating Th17 cells

Published:2009-12 Issue:2-3 Volume:47 Page:567-574
ISSN:0161-5890
Container-title:Molecular Immunology
language:en
Short-container-title:Molecular Immunology

Author:

Chen Shyi-Jou,Wang Yen-Lin,Kao Jen-Hsin,Wu Shu-Feng,Lo Wen-Tsung,Wu Chia-Cho,Tao Pao-Luh,Wang Chih-Chien,Chang Deh-Ming,Sytwu Huey-Kang

Funder

National Science Council (Taiwan, Republic of China)

TSGH-C98-18

C.Y. Foundation for Advancement of Education, Sciences, and Medicine

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference38 articles.

1. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells;Bettelli;Nature,2006

2. IL-4 is the key regulator in herpes simplex virus-based gene therapy of BALB/c experimental autoimmune encephalomyelitis;Broberg;Neurosci. Lett.,2004

3. Relapses and progression of disability in multiple sclerosis;Confavreux;N. Engl. J. Med.,2000

4. Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain;Cua;Nature,2003

5. Production of IL-1beta and IL-1Ra as risk factors for susceptibility and progression of relapse-onset multiple sclerosis;de Jong;J. Neuroimmunol.,2002

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