Loss of Cxcr4 in Endometriosis Reduces Proliferation and Lesion Number while Increasing Intraepithelial Lymphocyte Infiltration
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Pathology and Forensic Medicine
Reference49 articles.
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1. Maternal CXCR4 deletion results in placental defects and pregnancy loss mediated by immune dysregulation;JCI Insight;2023-11-08
2. METTL3-mediated m6A modification of SIRT1 mRNA inhibits progression of endometriosis by cellular senescence enhancing;Journal of Translational Medicine;2023-06-23
3. CXC chemokines influence immune surveillance in immunological disorders: Polycystic ovary syndrome and endometriosis;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2023-06
4. Dissecting the miR-451a-Mif Pathway in Endometriosis Pathophysiology Using a Syngeneic Mouse Model: Temporal Expression of Lesion Mif Receptors, Cd74 and Cxcr4;Biomedicines;2022-07-14
5. CPEB3, an RNA-Binding Protein, Modulates the Behavior of Endometriosis-Derived Stromal Cells via Regulating CXCL12;DNA and Cell Biology;2022-06-01
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