Preferential uncoupling by manganese of alpha adrenergic receptor mediated inhibition of adenylate cyclase in human platelets
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference12 articles.
1. Biochemical Properties of Hormone-Sensitive Adenylate Cyclase
2. Inhibition of platelet adenylate cyclase by epinephrine requires GTP
3. Regulation of human platelet adenylate cyclase by epinephrine, prostaglandin E1, and guanine nucleotides. Evidence for separate guanine nucleotide sites mediating stimulation and inhibition.
4. Heterogeneity of radioligand binding to alpha-adrenergic receptors. Analysis of guanine nucleotide regulation of agonist binding in relation to receptor subtypes.
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1. Adrenergic Receptors: Classification, ligand binding and molecular properties;Acta Medica Scandinavica;2009-04-24
2. Stable GDP analog-induced inactivation of Gi proteins promotes cardiac adenylyl cyclase inhibition by guanosine 5′-(βγ-imino) triphosphate and muscarinic acetylcholine receptor;Biochimica et Biophysica Acta (BBA) - Biomembranes;1996-06
3. GABAB receptors;Pharmacology & Therapeutics;1995-01
4. The role of Gi and the membrane-fluidizing agent benzyl alcohol in modulating the hysteretic activation of human platelet adenylate cyclase by guanylyl 5′-imidodiphosphate;Biochemical Journal;1993-05-01
5. Receptor-mediated supra-additive activation of guinea pig superior cervical ganglion adenylate cyclase: Role of Mn2+ ions and calmodulin;Neurochemical Research;1991-05
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