The vast complexity of primary open angle glaucoma: Disease genes, risks, molecular mechanisms and pathobiology

Author:

Janssen Sarah F.,Gorgels Theo G.M.F.,Ramdas Wishal D.,Klaver Caroline C.W.,van Duijn Cornelia M.,Jansonius Nomdo M.,Bergen Arthur A.B.

Funder

General Dutch Foundation

National Foundation

Foundation Blinden-Penning

Foundation Glaucoomfonds

Rotterdam Foundation

Professor Mulder Foundation

Publisher

Elsevier BV

Subject

Sensory Systems,Ophthalmology

Reference480 articles.

1. Glutathione S-transferase M1 and T1 polymorphisms in Arab glaucoma patients;Abu-Amero;Mol. Vis.,2008

2. Lack of association of SNP rs4236601 near CAV1 and CAV2 with POAG in a Saudi cohort;Abu-Amero;Mol. Vis.,2012

3. Primary role of CYP1B1 in Indian juvenile-onset POAG patients;Acharya;Mol. Vis.,2006

4. Complex genetics of glaucoma: defects in CYP1B1, and not MYOC, cause pathogenesis in an early-onset POAG patient with double variants at both loci;Acharya;J. Genet.,2008

5. Central corneal thickness of Caucasians, Chinese, Hispanics, Filipinos, African Americans, and Japanese in a glaucoma clinic;Aghaian;Ophthalmology,2004

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