The acute increases in vasomotor tone and blood pressure induced by carotid artery occlusion are modulated by platelet-activating factor (PAF) independently of nitric oxide release

Author:

Lopes-Martins Rodrigo A.B,Estato Vanessa,Araújo Cláudia V,Catelli Marcelo,Cordeiro Renato S.B,Tibiriçá Eduardo

Publisher

Elsevier BV

Subject

Pharmacology,Immunology

Reference59 articles.

1. Nitric oxide-endothelin-1 interaction in humans;Ahlborg;J Appl Physiol,1997

2. PAF antagonists block induction of nitric oxide synthase in cultured macrophages and vascular smooth muscle cells;Arthur;Clin Exp Pharmacol Physiol,1995

3. Acute hypertension after nitric oxide synthase inhibition is mediated primarily by increased endothelin vasoconstriction;Banting;J Hypertens,1996

4. Analysis of pulmonary and systemic vascular response to platelet activating factor in the cat;Bellan;Am J Physiol,1992

5. Acute circulatory collapse caused by platelet-activating factor (PAF-acether) in dogs;Bessin;Eur J Pharmacol,1983

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