Withdrawn: “Sulforaphane Restores Oxidative Stress Induced by Di-n-butylphthalate in Testicular Leydig Cells With Low Basal Reactive Oxygen Species Levels” [URL 84 (2014) 850 - 856]
Author:
Publisher
Elsevier BV
Subject
Urology
Reference22 articles.
1. Increasing prevalence of hypospadias associated with various perinatal risk factors in Chinese newborns;Sun;Urology,2009
2. Testicular dysgenesis syndrome: an increasingly common developmental disorder with environmental aspects;Skakkebaek;Hum Reprod,2001
3. Effects of in utero exposure to DI(n-Butyl) phthalate on development of male reproductive tracts in Sprague-Dawley rats;Kim;J Toxicol Environ Health A,2010
4. Study on developmental abnormalities in hypospadiac male rats induced by maternal exposure to di-n-butyl phthalate (DBP);Jiang;Toxicology,2007
5. Species-specific dibutyl phthalate fetal testis endocrine disruption correlates with inhibition of SREBP2-dependent gene expression pathways;Johnson;Toxicol Sci,2011
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2. The role of ANXA5 in DBP-induced oxidative stress through ERK/Nrf2 pathway;Environmental Toxicology and Pharmacology;2019-11
3. A strategy to validate a selection of human effect biomarkers using adverse outcome pathways: Proof of concept for phthalates and reproductive effects;Environmental Research;2019-08
4. Sulforaphane attenuates di-N-butylphthalate-induced reproductive damage in pubertal mice: Involvement of the Nrf2-antioxidant system;Environmental Toxicology;2017-03-15
5. Disruption of steroidogenesis: Cell models for mechanistic investigations and as screening tools;The Journal of Steroid Biochemistry and Molecular Biology;2016-04
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