The accumulation of assembly intermediates of the mitochondrial complex I matrix arm is reduced by limiting glucose uptake in a neuronal-like model of MELAS syndrome-Reference-Cited by-同舟云学术

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The accumulation of assembly intermediates of the mitochondrial complex I matrix arm is reduced by limiting glucose uptake in a neuronal-like model of MELAS syndrome

Published:2018-05 Issue:5 Volume:1864 Page:1596-1608
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Geffroy Guillaume,Benyahia Rayane,Frey Samuel,Desquiret-Dumas Valerie,Gueguen Naig,Bris Celine,Belal Sophie,Inisan Aurore,Renaud Aurelie,Chevrollier Arnaud,Henrion Daniel,Bonneau Dominique,Letournel Franck,Lenaers Guy,Reynier Pascal,Procaccio Vincent

Funder

Fondation pour la Recherche Médicale

Association Française contre les Myopathies

Région Pays de la Loire

Association contre les Maladies Mitochondriales

University and Hospital of Angers

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference60 articles.

1. Mitochondrial energetics and therapeutics;Wallace;Annu. Rev. Pathol.,2010

2. A mitochondrial paradigm of metabolic and degenerative diseases, aging, and cancer: a dawn for evolutionary medicine;Wallace;Annu. Rev. Genet.,2005

3. Why do we still have a maternally inherited mitochondrial DNA? Insights from evolutionary medicine;Wallace;Annu. Rev. Biochem.,2007

4. A mutation in the tRNA(Leu)(UUR) gene associated with the MELAS subgroup of mitochondrial encephalomyopathies;Goto;Nature,1990

5. Prevalence, segregation, and phenotype of the mitochondrial DNA 3243A>G mutation in children;Uusimaa;Ann. Neurol.,2007

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