Loss of USP22 alleviates cardiac hypertrophy induced by pressure overload through HiF1-α-TAK1 signaling pathway-Reference-Cited by-同舟云学术

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Loss of USP22 alleviates cardiac hypertrophy induced by pressure overload through HiF1-α-TAK1 signaling pathway

Published:2023-12 Issue:8 Volume:1869 Page:166813
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Jiang Yi-na,Yang Shou-xing,Guan Xuan,Chen Qiaoying,Zhao Lin,Yu Xiao-yu,Ren Fang-fang,Wu Shu-jie,Wu Lian-pin,Lai Teng-Fang,Li Lei

Funder

Natural Science Foundation of Zhejiang Province

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference36 articles.

1. Molecular pathways underlying cardiac remodeling during pathophysiological stimulation;Kehat;Circulation.,2010

2. Melatonin reduces cardiac remodeling and improves survival in rats with isoproterenol-induced heart failure;Simko;J. Pineal Res.,2014

3. Collagen remodeling of the pressure-overloaded, hypertrophied nonhuman primate myocardium;Weber;Circ. Res.,1988

4. Small-molecule therapies for cardiac hypertrophy: moving beneath the cell surface;McKinsey;Nat. Rev. Drug Discov.,2007

5. Mechanisms of deubiquitinase specificity and regulation;Mevissen;Annu. Rev. Biochem.,2017

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The role of deubiquitinases in cardiac disease;Expert Reviews in Molecular Medicine;2024

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