BRAF promotes anchorage-independent growth but inhibits anchorage-dependent growth in hTERT/Cdk4-Immortalized normal human bronchial epithelial cells-Reference-Cited by-同舟云学术

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BRAF promotes anchorage-independent growth but inhibits anchorage-dependent growth in hTERT/Cdk4-Immortalized normal human bronchial epithelial cells

Published:2024-06 Issue:1 Volume:439 Page:114057
ISSN:0014-4827
Container-title:Experimental Cell Research
language:en
Short-container-title:Experimental Cell Research

Author:

Muraki Nao^ORCID,Kawabe Nozomi,Ohashi Ayano,Umeda Kanna,Katsuda Masahito^ORCID,Tomatsu Aya,Yoshida Mikina^ORCID,Komeda Kazuki,Minna John D.,Tanaka Ichidai,Morise Masahiro,Matsushima Miyoko^ORCID,Matsui Yusuke,Kawabe Tsutomu^ORCID,Sato Mitsuo^ORCID

Funder

Ministry of Education, Culture, Sports, Science and Technology

Nagoya University

Iwadare Scholarship Foundation

Publisher

Elsevier BV

Reference35 articles.

1. Targeting the RAS/RAF/MAPK pathway for cancer therapy: from mechanism to clinical studies;Bahar;Signal Transduct. Targeted Ther.,2023

2. Targeted therapies for lung cancer patients with oncogenic driver molecular alterations;Tan;J. Clin. Oncol.,2022

3. Immortalization of human bronchial epithelial cells in the absence of viral oncoproteins;Ramirez;Cancer Res.,2004

4. Immortalized normal human lung epithelial cell models for studying lung cancer biology;Sato;Respir Investig,2020

5. Multiple oncogenic changes (K-RAS(V12), p53 knockdown, mutant EGFRs, p16 bypass, telomerase) are not sufficient to confer a full malignant phenotype on human bronchial epithelial cells;Sato;Cancer Res.,2006

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