Acquisition of Cabozantinib-Sensitive MET D1228N Mutation During Progression on Crizotinib in MET-Amplified Triple-Negative Breast Cancer
Author:
Funder
Abbvie
Genentech
Puma
Loxo Oncology
Gundersen Medical Foundation
Publisher
Elsevier BV
Subject
Cancer Research,Oncology
Reference24 articles.
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3. Responses to crizotinib can occur in high-level MET-amplified non-small cell lung cancer independent of MET exon 14 alterations;Caparica;J Thorac Oncol,2017
4. MET amplification identifies a small and aggressive subgroup of esophagogastric adenocarcinoma with evidence of responsiveness to crizotinib;Lennerz;J Clin Oncol,2011
5. Crizotinib achieves long-lasting disease control in advanced papillary renal-cell carcinoma type 1 patients with MET mutations or amplification. EORTC 90101 CREATE trial;Schoffski;Eur J Cancer,2017
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1. Integrative genomic analysis of drug resistance in MET exon 14 skipping lung cancer using patient-derived xenograft models;Frontiers in Oncology;2022-10-21
2. Foretinib can overcome common on-target resistance mutations after capmatinib/tepotinib treatment in NSCLCs with MET exon 14 skipping mutation;Journal of Hematology & Oncology;2022-06-11
3. Cationic liposomes co-deliver chemotherapeutics and siRNA for the treatment of breast cancer;European Journal of Medicinal Chemistry;2022-04
4. Cabozantinib/crizotinib;Reactions Weekly;2022-01
5. MET D1228N and D1246N are the Same Resistance Mutation in MET Exon 14 Skipping;The Oncologist;2021-08-17
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