Intron retention is a mechanism of erythropoietin regulation in brain cell models
Author:
Publisher
Elsevier BV
Subject
Genetics,General Medicine
Reference47 articles.
1. Adusumalli, S., Ngian, Z.K., Lin, W.Q., Benoukraf, T., Ong, C.T., 2019. Increased intron retention is a post-transcriptional signature associated with progressive aging and Alzheimer’s disease. Aging Cell 18, 1–13. 10.1111/acel.12928.
2. Alternatively spliced mRNAs encoding soluble isoforms of the erythropoietin receptor in murine cell lines and bone marrow;Barron;Gene,1994
3. EV-3, an endogenous human erythropoietin isoform with distinct functional relevance;Bonnas;Sci. Rep.,2017
4. Widespread intron retention in mammals functionally tunes transcriptomes;Braunschweig;Genome Res.,2014
5. Cytoplasmic intron sequence-retaining transcripts (CIRTs) can be dendritically targeted via ID element retrotransposons;Buckley;Neuron,2011
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