Interleukin-11-driven gastric tumourigenesis is independent of trans-signalling

Author:

Balic Jesse J.,Garbers Christoph,Rose-John Stefan,Yu Liang,Jenkins Brendan J.

Funder

National Health and Medical Research Council (NHMRC) of Australia

Victorian Government of Australia

Deutsche Forschungsgemeinschaft Bonn, Germany

German Research Foundation

Australian Government

NHMRC Senior Medical Research Fellowship

Publisher

Elsevier BV

Subject

Molecular Biology,Hematology,Biochemistry,Immunology,Immunology and Allergy

Reference15 articles.

1. Inflammation, atrophy, and gastric cancer;Fox;J. Clin. Invest.,2007

2. IL-6 and IL-11 as markers for tumor aggressiveness and prognosis in gastric adenocarcinoma patients without mutations in Gp130 subunits;Necula;J. Gastrointestin. Liver Dis.,2012

3. Identification of protein expression signatures associated with Helicobacter pylori infection and gastric adenocarcinoma using recombinant antibody microarrays;Ellmark;Mol. Cell Proteom.,2006

4. STAT3-driven upregulation of TLR2 promotes gastric tumorigenesis independent of tumor inflammation;Tye;Cancer Cell,2012

5. STAT3 and STAT1 mediate IL-11-dependent and inflammation-associated gastric tumorigenesis in gp130 receptor mutant mice;Ernst;J. Clin. Invest.,2008

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