IL-22 relieves hepatic ischemia-reperfusion injury by inhibiting mitochondrial apoptosis based on the activation of STAT3-Reference-Cited by-同舟云学术

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IL-22 relieves hepatic ischemia-reperfusion injury by inhibiting mitochondrial apoptosis based on the activation of STAT3

Published:2024-01 Issue: Volume:166 Page:106503
ISSN:1357-2725
Container-title:The International Journal of Biochemistry & Cell Biology
language:en
Short-container-title:The International Journal of Biochemistry & Cell Biology

Author:

Jiang Zhengchen,Li Wanzhen,Yu Shuna,Wang Xuyang,Jiang Hongxin,Bai Chen,Li Ming,Chu Fangfang,Jiang Jiying,Ma Xiaomin

Funder

National Natural Science Foundation of China-Shandong Joint Fund for Marine Science Research Centers

China Postdoctoral Science Foundation

Natural Science Foundation of Shandong Province

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Reference45 articles.

1. Mitochondria as playmakers of apoptosis, autophagy and senescence;Abate;Semin. Cell Dev. Biol.,2020

2. IL-22 protects against biliary ischemia-reperfusion injury after liver transplantation via activating STAT3 and reducing apoptosis and oxidative stress levels in vitro and in vivo;Bai;Oxid. Med. Cel.l Longev.,2022

3. IL-22 ameliorated cardiomyocyte apoptosis in cardiac ischemia/reperfusion injury by blocking mitochondrial membrane potential decrease, inhibiting ROS and cytochrome C;Che;Biochim. Biophys. Acta Mol. Basis Dis.,2021

4. Tripartite motif-containing 27 attenuates liver ischemia/reperfusion injury by suppressing transforming growth factor β-activated kinase 1 (TAK1) by TAK1 binding protein 2/3 degradation;Chen;Hepatology,2021

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