JMJD3 as an epigenetic regulator in development and disease
Author:
Funder
NIH
Cancer Prevention and Research Institution of Texas
Publisher
Elsevier BV
Subject
Cell Biology,Biochemistry
Reference97 articles.
1. The H3K27me3 demethylase JMJD3 contributes to the activation of the INK4A-ARF locus in response to oncogene- and stress-induced senescence;Agger;Genes Dev.,2009
2. Polycomb mediated epigenetic silencing and replication timing at the INK4a/ARF locus during senescence;Agherbi;PLoS ONE,2009
3. H3K27me3 regulates BMP activity in developing spinal cord;Akizu;Development (Cambridge, England),2010
4. The H3K27me3 demethylase, KDM6B, is induced by Epstein-Barr virus and over-expressed in Hodgkin's lymphoma;Anderton;Oncogene,2011
5. Chromatin dynamics during cellular reprogramming;Apostolou;Nature,2013
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