Differential regulation of CFTRΔF508 degradation by ubiquitin ligases gp78 and Hrd1
Author:
Funder
Scientific and Technological Research Council of Turkey
Turkish Academy of Science
Young Investigator Award
Publisher
Elsevier BV
Subject
Cell Biology,Biochemistry
Reference37 articles.
1. The role of a novel p97/valosin-containing protein-interacting motif of gp78 in endoplasmic reticulum-associated degradation;Ballar;J Biol Chem,2006
2. Identification of SVIP as an endogenous inhibitor of endoplasmic reticulum-associated degradation;Ballar;J Biol Chem,2007
3. The activity of a human endoplasmic reticulum-associated degradation E3, gp78, requires its Cue domain, RING finger, and an E2-binding site;Chen;Proc Natl Acad Sci U S A,2006
4. Arrest of CFTRDeltaF508 folding;Cyr;Nat Struct Mol Biol,2005
5. Altered chloride ion channel kinetics associated with the delta F508 cystic fibrosis mutation;Dalemans;Nature,1991
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