Effects of sclerostin on lipopolysaccharide-induced inflammatory phenotype in human odontoblasts and dental pulp cells
Author:
Funder
National Natural Science Foundation of China
Publisher
Elsevier BV
Subject
Cell Biology,Biochemistry
Reference59 articles.
1. TNF-alpha upregulates sclerostin expression in obese mice fed a high-fat diet;Baek;J. Cell. Physiol.,2014
2. Gingival crevicular fluid levels of sclerostin, Osteoprotegerin, and receptor activator of nuclear Factor-kappaB ligand in periodontitis;Balli;J. Periodontol.,2015
3. TLR4 mediates LPS-induced VEGF expression in odontoblasts;Botero;J. Endod.,2006
4. MAPK Signaling is required for LPS-induced VEGF in pulp stem cells;Botero;J. Dent. Res.,2010
5. Angiogenic properties of human dental pulp stem cells;Bronckaers;PLoS One,2013
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1. The Role of Cellular Metabolism in Maintaining the Function of the Dentine-Pulp Complex: A Narrative Review;Metabolites;2023-04-05
2. Modulators of Wnt Signaling Pathway Implied in Dentin Pulp Complex Engineering: A Literature Review;International Journal of Molecular Sciences;2022-09-13
3. Gingival Mesenchymal Stem Cells Metabolite Decreasing TRAP, NFATc1, and Sclerostin Expression in LPS-Associated Inflammatory Osteolysis In Vivo;European Journal of Dentistry;2022-06-21
4. Sclerostin is a promising therapeutic target for oral inflammation and regenerative dentistry;Journal of Translational Medicine;2022-05-13
5. Role of Lipopolysaccharide, Derived from Various Bacterial Species, in Pulpitis—A Systematic Review;Biomolecules;2022-01-15
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