JAK2-V617F is a negative regulation factor of SHIP1 protein and thus influences the AKT signaling pathway in patients with Myeloproliferative neoplasm (MPN)
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Biochemistry
Reference74 articles.
1. The impact of peripheral blood values and bone marrow findings on prognosis for patients with essential thrombocythemia and polycythemia vera;Abdulkarim;Eur. J. Haematol. 86,2011
2. Fyn and JAK2 mediate ras activation by reactive oxygen species;Abe;J. Biol. Chem.,1999
3. Inhibiting the Akt pathway in cancer treatment;Alexander;P T,2011
4. Rationale for targeting the PI3K/Akt/mTOR pathway in myeloproliferative neoplasms;Bartalucci;Clin. Lymphoma Myeloma Leuk.,2013
5. The t(4;22)(q12;q11) in atypical chronic myeloid leukaemia fuses BCR to PDGFRA;Baxter;Hum. Mol. Genet,2002
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2. Activated Src kinases downstream of BCR-ABL and Flt3 induces proteasomal degradation of SHIP1 by phosphorylation of tyrosine 1021;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2023-06
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