Production of endogenous nitric oxide and activation of soluble guanylate cyclase are required for N-methyl-D-aspartate-produced facilitation of the nociceptive tail-flick reflex

Author:

Meller Stephen T.,Dykstra Carrie,Gebhart G.F.

Publisher

Elsevier BV

Subject

Pharmacology

Reference10 articles.

1. Nociceptive action of excitatory amino acids in the mouse: effects of spinally administered opioids, phencyclidine and sigma agonists;Aanonsen;J. Pharmacol. Exp. Ther.,1987

2. The behavioral effects of an N-methylaspartate receptor antagonist following application to the lumbar spinal cord of conscious rats;Cahusac;Neuropharmacology,1984

3. MK-801 blocks the development of thermal hyperalgesia in a rat model of experimental painful neuropathy;Davar;Brain Res.,1991

4. Antagonism at the glycine site on the NMDA receptor reduces spinal nociception in the rat;Dickenson;Neurosci. Lett.,1991

5. Peripheral analgesia and activation of the nitric oxide-cyclic GMP pathway;Duarte;Eur. J. Pharmacol.,1990

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