Distribution of oligodendrocyte loss and mitochondrial toxicity in the cuprizone-induced experimental demyelination model

Author:

Acs P.,Selak M.A.,Komoly S.,Kalman B.

Funder

Veterans Administration

NMSS

Serono Foundation

Hungarian Neuroimaging Foundation

Publisher

Elsevier BV

Subject

Clinical Neurology,Neurology,Immunology,Immunology and Allergy

Reference20 articles.

1. Preferential loss of myelin-associated glycoprotein reflects hypoxia-like white matter damage in stroke and inflammatory brain diseases;Aboul-Enein;J. Neuropathol. Exp. Neurol.,2003

2. Selective ultrastructural vulnerability in the cuprizone-induced experimental demyelination;Acs;Ideggyogy Sz.,2012

3. The use of acetylated ferricytochrome c for the detection of superoxide radical produced in biological membranes;Azzi;Biochem. Biophys. Res. Commun.,1975

4. Relapsing and remitting multiple sclerosis: pathology of the newly forming lesion;Barnett;Ann. Neurol.,2004

5. Oligodendrocytes: biology and pathology;Bradl;Acta Neuropathol,2010

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