Macrophage-specific nanotechnology-driven CD163 overexpression in human macrophages results in an M2 phenotype under inflammatory conditions

Author:

Alvarado-Vazquez Perla Abigail,Bernal Laura,Paige Candler A.,Grosick Rachel L.,Moracho Vilrriales Carolina,Ferreira David Wilson,Ulecia-Morón Cristina,Romero-Sandoval E. Alfonso

Publisher

Elsevier BV

Subject

Hematology,Immunology,Immunology and Allergy

Reference61 articles.

1. CD163 interacts with TWEAK to regulate tissue regeneration after ischaemic injury;Akahori;Nat. Commun.,2015

2. CD163 and CD206 expression does not correlate with tolerance and cytokine production in LPS-tolerant human monocytes;Alves-Januzzi;Cytometry B Clin. Cytom.,2015

3. Evaluation of a nanotechnology-based approach to induce gene-expression in human THP-1 macrophages under inflammatory conditions;Bernal;Immunobiology,2016

4. THP-1 cells as a model for human monocytes;Bosshart;Ann. Transl. Med.,2016

5. A previously unrecognized protein−protein interaction between TWEAK and CD163: potential biological implications;Bover;J. Immunol.,2007

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