Aph-1, Pen-2, and Nicastrin with Presenilin Generate an Active γ-Secretase Complex
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference22 articles.
1. A Cell Biological Perspective on Alzheimer's Disease
2. Human presenilin-1, but not familial Alzheimer’s disease (FAD) mutants, facilitate Caenorhabditis elegans Notch signalling independently of proteolytic processing
3. Deficiency of presenilin-1 inhibits the normal cleavage of amyloid precursor protein
4. Mice lacking both presenilin genes exhibitearlyembryonic patterningdefects
5. Presenilin and nicastrin regulate each other and determine amyloid -peptide production via complex formation
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