Involvement of ER Stress in Dysmyelination of Pelizaeus-Merzbacher Disease with PLP1 Missense Mutations Shown by iPSC-Derived Oligodendrocytes-Reference-Cited by-同舟云学术

Involvement of ER Stress in Dysmyelination of Pelizaeus-Merzbacher Disease with PLP1 Missense Mutations Shown by iPSC-Derived Oligodendrocytes

Published:2014-05 Issue:5 Volume:2 Page:648-661
ISSN:2213-6711
Container-title:Stem Cell Reports
language:en
Short-container-title:Stem Cell Reports

Author:

Numasawa-Kuroiwa Yuko,Okada Yohei,Shibata Shinsuke,Kishi Noriyuki,Akamatsu Wado,Shoji Masanobu,Nakanishi Atsushi,Oyama Manabu,Osaka Hitoshi,Inoue Ken,Takahashi Kazutoshi,Yamanaka Shinya,Kosaki Kenjiro,Takahashi Takao,Okano Hideyuki

Funder

Ministry of Education, Culture

Ministry of Education, Culture, Sports, Science and Technology of Japan

MEXT

Kanrinmaru-Project at Keio University

JST-CIRM

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Genetics,Biochemistry

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3. A cellular mechanism governing the severity of Pelizaeus-Merzbacher disease;Gow;Nat. Genet.,1996

4. Many naturally occurring mutations of myelin proteolipid protein impair its intracellular transport;Gow;J. Neurosci. Res.,1994

5. Conservation of topology, but not conformation, of the proteolipid proteins of the myelin sheath;Gow;J. Neurosci.,1997

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