Cellular Sites and Mechanisms Linking Reduction of Dipeptidyl Peptidase-4 Activity to Control of Incretin Hormone Action and Glucose Homeostasis

Author:

Mulvihill Erin E.,Varin Elodie M.,Gladanac Bojana,Campbell Jonathan E.,Ussher John R.,Baggio Laurie L.,Yusta Bernardo,Ayala Jennifer,Burmeister Melissa A.,Matthews Dianne,Bang K.W. Annie,Ayala Julio E.,Drucker Daniel J.

Funder

Canadian Institutes of Health Research

Canadian Diabetes Association

Alberta Innovates-Health Solutions

Banting and Best Diabetes Centre

University of Toronto

Canada Research Chair in Regulatory Peptides

Banting and Best Diabetes Centre-Novo Nordisk Chair in Incretin Biology

Novo Nordisk

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference37 articles.

1. Insulin action in the double incretin receptor knockout mouse;Ayala;Diabetes,2008

2. Glucagon-like peptide-1 receptor knockout mice are protected from high-fat diet-induced insulin resistance;Ayala;Endocrinology,2010

3. GLP-1-based strategies: a physiological analysis of differential mode of action;Burcelin;Physiology (Bethesda),2014

4. Pharmacology, physiology, and mechanisms of incretin hormone action;Campbell;Cell Metab.,2013

5. Mice lacking dipeptidyl peptidase IV are protected against obesity and insulin resistance;Conarello;Proc. Natl. Acad. Sci. USA,2003

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