Colonic Pro-inflammatory Macrophages Cause Insulin Resistance in an Intestinal Ccl2/Ccr2-Dependent Manner-Reference-Cited by-同舟云学术

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Colonic Pro-inflammatory Macrophages Cause Insulin Resistance in an Intestinal Ccl2/Ccr2-Dependent Manner

Published:2016-08 Issue:2 Volume:24 Page:295-310
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Kawano Yoshinaga,Nakae Jun,Watanabe Nobuyuki,Kikuchi Tetsuhiro,Tateya Sanshiro,Tamori Yoshikazu,Kaneko Mari,Abe Takaya,Onodera Masafumi,Itoh Hiroshi

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference55 articles.

1. IL-1beta causes an increase in intestinal epithelial tight junction permeability;Al-Sadi;J. Immunol.,2007

2. The biology of innate lymphoid cells;Artis;Nature,2015

3. Intestinal macrophages—specialised adaptation to a unique environment;Bain;Eur. J. Immunol.,2011

4. Constant replenishment from circulating monocytes maintains the macrophage pool in the intestine of adult mice;Bain;Nat. Immunol.,2014

5. CCR2 deficiency leads to increased eosinophils, alternative macrophage activation, and type 2 cytokine expression in adipose tissue;Bolus;J. Leukoc. Biol.,2015

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