CSE1L promotes nuclear accumulation of transcriptional coactivator TAZ and enhances invasiveness of human cancer cells
Author:
Funder
Japan Society for the Promotion of Science
Daiichi-Sankyo
Mitsubishi Foundation
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference36 articles.
1. TAZ: A novel transcriptional co-activator regulated by interactions with 14-3-3 and PDZ domain proteins;Kanai;EMBO J.,2000
2. TAZ promotes cell proliferation and epithelial-mesenchymal transition and is inhibited by the hippo pathway;Lei;Mol. Cell. Biol.,2008
3. The hippo tumor pathway promotes TAZ degradation by phosphorylating a phosphodegron and recruiting the SCF{beta}-TrCP E3 ligase;Liu;J. Biol. Chem.,2010
4. The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4;Huang;EMBO J.,1998
5. Molecular pathways: YAP and TAZ take center stage in organ growth and tumorigenesis;Piccolo;Clin. Cancer Res.,2013
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