The CCL2/CCL7/CCL12/CCR2 pathway is substantially and persistently upregulated in mice after traumatic brain injury, and CCL2 modulates the complement system in microglia-Reference-Cited by-同舟云学术

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The CCL2/CCL7/CCL12/CCR2 pathway is substantially and persistently upregulated in mice after traumatic brain injury, and CCL2 modulates the complement system in microglia

Published:2020-12 Issue: Volume:54 Page:101671
ISSN:0890-8508
Container-title:Molecular and Cellular Probes
language:en
Short-container-title:Molecular and Cellular Probes

Author:

Popiolek-Barczyk Katarzyna,Ciechanowska Agata,Ciapała Katarzyna,Pawlik Katarzyna,Oggioni Marco^ORCID,Mercurio Domenico^ORCID,De Simoni Maria-Grazia,Mika Joanna

Funder

ERA-NET-NEURON

JTC 2016: LEAP

NEURON9-FP-044

National Centre for Research and Development (Narodowe Centrum Badań i Rozwoju, ERA-NET NEURON-Cofund/1/LEAP/15/17), Poland

Italian Ministry of Health (Ministero della Salute), Italy

Maj Institute of Pharmacology

Polish Academy of Sciences

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference93 articles.

1. Changes in macrophage inflammatory protein-1 (MIP-1) family members expression induced by traumatic brain injury in mice;Ciechanowska;Immunobiology,2020

2. Fractalkine receptor deficiency is associated with early protection but late worsening of outcome following brain trauma in mice;Zanier;J. Neurotrauma,2016

3. A three-day consecutive fingolimod administration improves neurological functions and modulates multiple immune responses of CCI mice;Gao;Mol. Neurobiol.,2017

4. Novel mGluR 5 positive allosteric modulator improves functional recovery, attenuates neurodegeneration, and alters microglial polarization after experimental traumatic brain injury;Loane;Neurotherapeutics,2014

5. Inflammation after trauma: microglial activation and traumatic brain injury;Ramlackhansingh;Neurol.,2011

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