Endoplasmic reticulum stress promotes oxidative stress, inflammation, and apoptosis: A novel mechanism of citrinin-induced renal injury and dysfunction-Reference-Cited by-同舟云学术

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Endoplasmic reticulum stress promotes oxidative stress, inflammation, and apoptosis: A novel mechanism of citrinin-induced renal injury and dysfunction

Published:2024-10 Issue: Volume:284 Page:116946
ISSN:0147-6513
Container-title:Ecotoxicology and Environmental Safety
language:en
Short-container-title:Ecotoxicology and Environmental Safety

Author:

Wang Yongkang,Li Yuanyuan,Wu You,Wu Aoao,Xiao Bo,Liu Xiaofang,Zhang Qike,Feng Yiya,Yuan Zhihang,Yi Jine,Wu Jing,Yang Chenglin

Funder

Major Science and Technology Projects in Yunnan Province

Yunnan Provincial Science and Technology Department

National Natural Science Foundation of China

Publisher

Elsevier BV

Reference36 articles.

1. Hypoxia, endoplasmic reticulum stress and chemoresistance: dangerous liaisons;Akman;J. Exp. Clin. Cancer Res.,2021

2. Endoplasmic reticulum stress;Bánhegyi;Ann. N. Y. Acad. Sci.,2007

3. Sodium 4-phenylbutyrate treatment protects against renal injury in NZBWF1 mice;Bonnemaison;Clin. Sci.,2019

4. A molecular web: endoplasmic reticulum stress, inflammation, and oxidative stress;Chaudhari;Front. Cell Neurosci.,2014

5. Transcriptome study reveals apoptosis of porcine kidney cells induced by fumonisin B1 via TNF signalling pathway;Chen;Food Chem. Toxicol.,2020

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