Fine particulate matter (PM2.5) induces the stem cell-like properties of hepatocellular carcinoma by activating ROS/Nrf2/Keap1-mediated autophagy

Author:

Li Jiujiu,Jiang Haoqi,Zhu Yu,Ma Zijian,Li Bin,Dong Jun,Xiao Changchun,Hu Anla

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health,Pollution,General Medicine

Reference60 articles.

1. Nrf2-Keap1 signaling in oxidative and reductive stress;Bellezza;Biochim. Biophys. Acta Mol. Cell Res.,2018

2. TNF and ROS crosstalk in inflammation;Blaser;Trends Cell Biol.,2016

3. Interleukin-37 relieves PM2.5-triggered lung injury by inhibiting autophagy through the AKT/mTOR signaling pathway in vivo and in vitro;Cao;Ecotoxicol. Environ. Saf.,2023

4. Long-term PM(2.5) exposure increases the risk of non-small cell lung cancer (NSCLC) progression by enhancing interleukin-17a (IL-17a)-regulated proliferation and metastasis;Chao;Aging (Albany NY),2020

5. Integrated machine learning and bioinformatic analyses constructed a novel stemness-related classifier to predict prognosis and immunotherapy responses for hepatocellular carcinoma patients;Chen;Int. J. Biol. Sci.,2022

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