Long non-coding MIAT mediates high glucose-induced renal tubular epithelial injury-Reference-Cited by-同舟云学术

Long non-coding MIAT mediates high glucose-induced renal tubular epithelial injury

Published:2015-12 Issue:4 Volume:468 Page:726-732
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Zhou Ling,Xu De-yu,Sha Wen-gang,Shen Lei,Lu Guo-yuan,Yin Xia

Funder

Scientific and Technologic Development Project of Suzhou

Colleges Natural science research project of Jiangsu Province

Jiangsu Province Science Foundation of Youth Fund Project

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference23 articles.

1. Elevated urinary connective tissue growth factor in diabetic nephropathy is caused by local production and tubular dysfunction;Gerritsen;J. Diabetes Res.,2015

2. Tubular overexpression of gremlin in transgenic mice aggravates renal damage in diabetic nephropathy;Marchant;Am. J. Physiol. Ren. Physiol.,2015

3. Stimulatory effect of insulin on renal proximal tubule sodium transport is preserved in type 2 diabetes with nephropathy;Nakamura;Biochem. Biophys. Res. Commun.,2015

4. CXCR4 promotes renal tubular cell survival in male diabetic rats: implications for ligand inactivation in the human kidney;Siddiqi;Endocrinology,2015

5. PKCdelta promotes high glucose induced renal tubular oxidative damage via regulating activation and translocation of p66Shc;Song;Oxidative Med. Cell. Longev.,2014

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