Suppressed hepatocyte proliferation via a ROS-HNE-P21 pathway is associated with nicotine- and cotinine-enhanced alcoholic fatty liver in mice-Reference-Cited by-同舟云学术

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Suppressed hepatocyte proliferation via a ROS-HNE-P21 pathway is associated with nicotine- and cotinine-enhanced alcoholic fatty liver in mice

Published:2019-04 Issue:1 Volume:512 Page:119-124
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Chen Xue,Wang Kesheng,Cederbaum Arthur I.,Lu Yongke

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference39 articles.

1. Molecular mechanisms of alcoholic fatty liver;Purohit;Alcohol Clin. Exp. Res.,2009

2. Histologic spectrum of alcoholic liver disease;MacSween;Semin. Liver Dis.,1986

3. Molecular mechanisms of alcoholic liver disease: innate immunity and cytokines;Miller;Alcohol Clin. Exp. Res.,2011

4. Metabolism of alcohol;Lieber;Clin. Liver Dis.,2005

5. CYP2E1 and oxidative liver injury by alcohol;Lu;Free Radic. Biol. Med.,2008

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1. RNA-Seq Analysis of Protection against Chronic Alcohol Liver Injury by Rosa roxburghii Fruit Juice (Cili) in Mice;Nutrients;2022-05-09

2. PPARα agonist WY-14,643 enhances ethanol metabolism in mice: Role of catalase;Free Radical Biology and Medicine;2021-06

3. UVA Induced Oxidative Stress Was Inhibited by Paeoniflorin/Nrf2 Signaling or PLIN2;Frontiers in Pharmacology;2020-05-15

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