Novel DLX3 variant identified in a family with tricho-dento-osseous syndrome-Reference-Cited by-同舟云学术

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Novel DLX3 variant identified in a family with tricho-dento-osseous syndrome

Published:2022-09 Issue: Volume:141 Page:105479
ISSN:0003-9969
Container-title:Archives of Oral Biology
language:en
Short-container-title:Archives of Oral Biology

Author:

Liu Haochen,Wang Yue,Liu Hangbo,Yu Miao,Zheng Jinglei,Feng Hailan,Liu Yang,Han Dong

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Cell Biology,General Dentistry,General Medicine,Otorhinolaryngology

Reference31 articles.

1. Homeodomain proteins in action: Similar DNA binding preferences, highly variable connectivity;Bobola;Current Opinion in Genetics & Development,2017

2. In vivo impact of a 4 bp deletion mutation in the DLX3 gene on bone development;Choi;Developmental Biology,2009

3. A 4 bp deletion mutation in DLX3 enhances osteoblastic differentiation and bone formation in vitro;Choi;Bone,2008

4. Transcriptional activation by the homeodomain protein distal-less 3;Feledy;Nucleic Acids Research,1999

5. Increased bone density associated with DLX3 mutation in the tricho-dento-osseous syndrome;Haldeman;Bone,2004

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Fixed prosthodontic rehabilitation using a facially driven fully digital workflow of a patient with syndromic amelogenesis imperfecta associated with a rare form of ectodermal dysplasia, tricho-dento-osseous (TDO) syndrome;The Journal of Prosthetic Dentistry;2024-09

2. A novel DLX3 mutation causes tricho-dento-osseous syndrome with abnormal enamel structure and formation;Archives of Oral Biology;2024-01

3. Loss of Bmp2 impairs odontogenesis via dysregulating pAkt/pErk/GCN5/Dlx3/Sp7;2023-09-19

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