The RECQL4 protein, deficient in Rothmund–Thomson syndrome is active on telomeric D-loops containing DNA metabolism blocking lesions-Reference-Cited by-同舟云学术

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The RECQL4 protein, deficient in Rothmund–Thomson syndrome is active on telomeric D-loops containing DNA metabolism blocking lesions

Published:2013-07 Issue:7 Volume:12 Page:518-528
ISSN:1568-7864
Container-title:DNA Repair
language:en
Short-container-title:DNA Repair

Author:

Ferrarelli Leslie K.,Popuri Venkateswarlu,Ghosh Avik K.,Tadokoro Takashi,Canugovi Chandrika,Hsu Joseph K.,Croteau Deborah L.,Bohr Vilhelm A.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference67 articles.

1. Mitochondrial dysfunction leads to telomere attrition and genomic instability

2. A continuous correlation between oxidative stress and telomere shortening in fibroblasts

3. Telomere length mediates the effects of telomerase on the cellular response to genotoxic stress

4. Characterization of Oxidative Guanine Damage and Repair in Mammalian Telomeres

5. Aging and Genome Maintenance: Lessons from the Mouse?

Cited by 25 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Evolution and Functions of Telomeres;Function and Evolution of Repeated DNA Sequences;2023-12-29

2. Rothmund-Thomson syndrome, a disorder far from solved;Frontiers in Aging;2023-11-10

3. Role and Regulation of the RECQL4 Family during Genomic Integrity Maintenance;Genes;2021-11-29

4. Molecular Mechanisms of the RECQ4 Pathogenic Mutations;Frontiers in Molecular Biosciences;2021-11-18

5. Fanconi anemia-independent DNA inter-strand crosslink repair in eukaryotes;Progress in Biophysics and Molecular Biology;2020-12

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