CaM Kinase: Still Inspiring at 40
Author:
Funder
NIH
Publisher
Elsevier BV
Subject
General Neuroscience
Reference162 articles.
1. CaMKII in myocardial hypertrophy and heart failure;Anderson;J. Mol. Cell. Cardiol.,2011
2. Rapid dispersion of SynGAP from synaptic spines triggers AMPA receptor insertion and spine enlargement during LTP;Araki;Neuron,2015
3. Enzymatic activity of CaMKII is not required for its interaction with the glutamate receptor subunit GluN2B;Barcomb;Mol. Pharmacol.,2013
4. Autonomous CaMKII requires further stimulation by Ca2+/calmodulin for enhancing synaptic strength;Barcomb;FASEB J.,2014
5. The CaMKII/GluN2B protein interaction maintains synaptic strength;Barcomb;J. Biol. Chem.,2016
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1. Maternal deprivation causes CaMKII downregulation and modulates glutamate, norepinephrine and serotonin in limbic brain areas in a rat model of single prolonged stress;Journal of Affective Disorders;2024-03
2. Remote Ischemia Postconditioning Mitigates Hippocampal Neuron Impairment by Modulating Cav1.2-CaMKIIα-Aromatase Signaling After Global Cerebral Ischemia in Ovariectomized Rats;Molecular Neurobiology;2024-02-06
3. Neuropharmacological Evidence Implicating Drug-Induced Glutamate Receptor Dysfunction in Affective and Cognitive Sequelae of Subchronic Methamphetamine Self-Administration in Mice;International Journal of Molecular Sciences;2024-02-05
4. Enhanced capacity for CaMKII signaling mitigates calcium release related contractile fatigue with high intensity exercise;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2024-02
5. Contiguity in perception: origins in cellular associative computations;Trends in Neurosciences;2024-02
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