Molecular Substrates of Altered Axonal Growth and Brain Connectivity in a Mouse Model of Schizophrenia-Reference-Cited by-同舟云学术

Molecular Substrates of Altered Axonal Growth and Brain Connectivity in a Mouse Model of Schizophrenia

Published:2015-05 Issue:3 Volume:86 Page:680-695
ISSN:0896-6273
Container-title:Neuron
language:en
Short-container-title:Neuron

Author:

Mukai Jun,Tamura Makoto,Fénelon Karine,Rosen Andrew M.,Spellman Timothy J.,Kang Rujun,MacDermott Amy B.,Karayiorgou Maria,Gordon Joshua A.,Gogos Joseph A.

Publisher

Elsevier BV

Subject

General Neuroscience

Reference43 articles.

1. A signaling pathway AKTing up in schizophrenia;Arguello;J. Clin. Invest.,2008

2. Genetic and cognitive windows into circuit mechanisms of psychiatric disease;Arguello;Trends Neurosci.,2012

3. Neuroanatomy in adolescents and young adults with 22q11 deletion syndrome: comparison to an IQ-matched group;Baker;Neuroimage,2011

4. The neurocognitive phenotype of the 22q11.2 deletion syndrome: selective deficit in visual-spatial memory;Bearden;J. Clin. Exp. Neuropsychol.,2001

5. Generalized and specific cognitive performance in clinical high-risk cohorts: a review highlighting potential vulnerability markers for psychosis;Brewer;Schizophr. Bull.,2006

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