Phospholipase A2 products retain a neuron specific γ isoform of PKC on the plasma membrane through the C1 domain—a molecular mechanism for sustained enzyme activity
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Cellular and Molecular Neuroscience
Cited by 13 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. cPKCγ Inhibits Caspase-9-Initiated Neuronal Apoptosis in an Ischemia Reperfusion Model In Vitro Through p38 MAPK-p90RSK-Bad Pathway;Neurochemical Research;2022-09-24
2. cPKCγ ameliorates ischemic injury in cultured neurons exposed to oxygen glucose deprivation/reoxygenation by inhibiting ferroptosis;Neuroscience Research;2022-08
3. Downregulation of miR-338-3p alleviates neuronal ischemic injury by decreasing cPKCγ-Mediated autophagy through the Akt/mTOR pathway;Neurochemistry International;2022-03
4. Depression of Type I Diacylglycerol Kinases in Pancreatic β-Cells From Male Mice Results in Impaired Insulin Secretion;Endocrinology;2013-11-01
5. Polymeric Nanoparticles Containing Conjugated Phospholipase A2 for Nonviral Gene Delivery;Molecular Pharmaceutics;2010-06-08
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