Pharmacologic modeling of primary mitochondrial respiratory chain dysfunction in zebrafish-Reference-Cited by-同舟云学术

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Pharmacologic modeling of primary mitochondrial respiratory chain dysfunction in zebrafish

Published:2018-07 Issue: Volume:117 Page:23-34
ISSN:0197-0186
Container-title:Neurochemistry International
language:en
Short-container-title:Neurochemistry International

Author:

Byrnes James,Ganetzky Rebecca,Lightfoot Richard,Tzeng Michael^ORCID,Nakamaru-Ogiso Eiko,Seiler Christoph,Falk Marni J.^ORCID

Funder

National Institutes of Health

Will Woleben Family Research Fund

Jaxson Flynt Family Research Fund

Children's Hospital of Philadelphia Research Institute Genes, Genomes, and Pediatric Disease (GGPD) research affinity group

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience

Reference50 articles.

1. Mitochondrial metabolism in normal, myeloid, and erythroid hyperplastic rabbit bone marrow: effect of chloramphenicol;Abou-Khalil;Am. J. Hematol.,1980

2. Redesign of genetically encoded biosensors for monitoring mitochondrial redox status in a broad range of model eukaryotes;Albrecht;J. Biomol. Screen,2014

3. Teratological effects of a panel of sixty water-soluble toxicants on zebrafish development;Ali;Zebrafish,2014

4. Large-scale assessment of the zebrafish embryo as a possible predictive model in toxicity testing;Ali;PLoS One,2011

5. Treatment of mitochondrial disorders;Avula;Curr. Treat. Options Neurol.,2014

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