Mutant ubiquitin decreases amyloid β plaque formation in a transgenic mouse model of Alzheimer’s disease-Reference-Cited by-同舟云学术

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Mutant ubiquitin decreases amyloid β plaque formation in a transgenic mouse model of Alzheimer’s disease

Published:2012-10 Issue:5 Volume:61 Page:739-748
ISSN:0197-0186
Container-title:Neurochemistry International
language:en
Short-container-title:Neurochemistry International

Author:

van Tijn Paula,Dennissen Frank J.A.,Gentier Romina J.G.,Hobo Barbara,Hermes Denise,Steinbusch Harry W.M.,Van Leeuwen Fred W.,Fischer David F.

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience

Reference47 articles.

1. Beta-amyloid accumulation impairs multivesicular body sorting by inhibiting the ubiquitin–proteasome system;Almeida;J. Neurosci.,2006

2. BACE1 is the major beta-secretase for generation of Abeta peptides by neurons;Cai;Nat. Neurosci.,2001

3. Review: Unchained maladie - a reassessment of the role of Ubb(+1) -capped polyubiquitin chains in Alzheimer’s disease;Chadwick;Neuropathol. Appl. Neurobiol.,2012

4. Alzheimer’s disease: correlation of the suppression of beta-amyloid peptide secretion from cultured cells with inhibition of the chymotrypsin-like activity of the proteasome;Christie;J. Neurochem.,1999

5. The ubiquitin proteasome system in neurodegenerative diseases: sometimes the chicken, sometimes the egg;Ciechanover;Neuron,2003

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