A novel association between sinus node dysfunction and an SCN5A variant presenting as persistent symptomatic bradycardia in a young adult-Reference-Cited by-同舟云学术

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A novel association between sinus node dysfunction and an SCN5A variant presenting as persistent symptomatic bradycardia in a young adult

Published:2023-09 Issue:9 Volume:9 Page:662-666
ISSN:2214-0271
Container-title:HeartRhythm Case Reports
language:en
Short-container-title:HeartRhythm Case Reports

Author:

Kisling Adam^ORCID,Klusewitz Seth,Pagani Austin,Haigney Mark,Needleman Matthew

Funder

Uniformed Services University of the Health Sciences

Defense Health Agency

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference15 articles.

1. Congenital sick sinus syndrome caused by recessive mutations in the cardiac sodium channel gene (Scn5a);Benson;J Clin Invest,2003

2. A truncating SCN5A mutation combined with genetic variability causes sick sinus syndrome and early atrial fibrillation;Ziyadeh-Isleem;Heart Rhythm,2014

3. Genotype–phenotype relationship and risk stratification in loss of function SCN5A mutation carriers;Robyns;Ann Noninvasive Electrocardiol,2018

4. The cardiac sodium channel gene SCN5A and its gene product NaV1.5: role in physiology and pathophysiology;Veerman;Gene,2015

5. Impaired neuronal sodium channels cause intranodal conduction failure and reentrant arrhythmias in human sinoatrial node;Li;Nat Commun,2020

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