Pressure and endothelial coculture upregulate myocytic Fas–FasL pathway and induce apoptosis by way of direct and paracrine mechanisms
Author:
Publisher
Elsevier BV
Subject
General Medicine,Surgery
Reference22 articles.
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2. Chronic high pressure potentiates the antiproliferative effect and abolishes contractile phenotypic changes caused by endothelial cells in cocultured smooth muscle cells;Vouyouka;J Surg Res,2003
3. Ambient pulsatile pressure modulates endothelial cell proliferation;Vouyouka;J Mol Cell Cardiol,1998
4. Apoptosis of vascular smooth muscle cells is induced by Fas ligand derived from endothelial cells;Imanishi;Jpn Circ J,2001
5. Caspase-3-mediated processing of poly(ADP-ribose) glycohydrolase during apoptosis;Affar;J Biol Chem,2001
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3. Regulation of Vascular Smooth Muscle Cell Turnover by Endothelial Cell–Secreted MicroRNA-126;Circulation Research;2013-06-21
4. Ambient pressure upregulates nitric oxide synthase in a phosphorylated-extracellular regulated kinase– and protein kinase C–dependent manner;Journal of Vascular Surgery;2006-11
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