AP-2γ Induces p21 Expression, Arrests Cell Cycle, Inhibits the Tumor Growth of Human Carcinoma Cells
Author:
Publisher
Elsevier BV
Subject
Cancer Research
Reference44 articles.
1. Positive and negative regulation of transcription in vitro: enhancer-binding protein AP-2 is inhibited by SV40T antigen;Mitchell;Cell,1987
2. Cloning and expression of AP-2, a cell-type-specific transcription factor that activates inducible enhancer elements;Williams;Genes Dev,1988
3. Cloning and characterization of a second AP-2 transcription factor: AP-2 beta;Moser;Development,1995
4. AP-2.2, a novel gene related to AP-2, is expressed in the forebrain, limbs and face during mouse embryogenesis;Chazaud;Mech Dev,1996
5. Cloning and characterization of a novel mouse AP-2 transcription factor, AP-2delta, with unique DNA binding and transactivation properties;Zhao;J Biol Chem,2001
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