Effects of chromogranin A deficiency and excess in vivo: biphasic blood pressure and catecholamine responses-Reference-Cited by-同舟云学术

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Effects of chromogranin A deficiency and excess in vivo: biphasic blood pressure and catecholamine responses

Published:2010-04 Issue:4 Volume:28 Page:817-825
ISSN:0263-6352
Container-title:Journal of Hypertension
language:en
Short-container-title:

Author:

Vaingankar Sucheta M,Li Ying,Biswas Nilima,Gayen Jiaur,Choksi Sonia,Rao Fangwen,Ziegler Michael G,Mahata Sushil K,O'Connor Daniel T

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine,Physiology,Internal Medicine

Reference41 articles.

1. Chromogranin A, the major catecholamine storage vesicle soluble protein. Multiple size forms, subcellular storage, and regional distribution in chromaffin and nervous tissue elucidated by radioimmunoassay;O'Connor;J Biol Chem,1984

2. The chromogranin-secretogranin family;Taupenot;N Engl J Med,2003

3. Hypertension from targeted ablation of chromogranin A can be rescued by the human ortholog;Mahapatra;J Clin Invest,2005

4. Chromogranin A: implications for hypertension;O'Connor;J Hypertens Suppl,1984

5. Chromogranin A. Storage and release in hypertension;Takiyyuddin;Hypertension,1990

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1. Prevention of vascular calcification by the endogenous chromogranin A-derived mediator that inhibits osteogenic transdifferentiation;Basic Research in Cardiology;2021-10-13

2. The chromogranin A 1‐373 fragment reveals how a single change in the protein sequence exerts strong cardioregulatory effects by engaging neuropilin‐1;Acta Physiologica;2020-11

3. Catestatin reverses the hypertrophic effects of norepinephrine in H9c2 cardiac myoblasts by modulating the adrenergic signaling;Molecular and Cellular Biochemistry;2019-12-02

4. CATESTATIN AS A NOVEL MARKER OF CARDIOVASCULAR RISK IN SYSTEMIC HYPERTENSION;Russian Journal of Cardiology;2018-05-09

5. Mice overexpressing chromogranin A display hypergranulogenic adrenal glands with attenuated ATP levels contributing to the hypertensive phenotype;Journal of Hypertension;2018-05

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