Pathophysiology of rheumatoid arthritis
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Rheumatology
Reference54 articles.
1. HLA-DR1001 presents ‘altered-self’ peptides derived from joint-associated proteins by accepting citrulline in three of its binding pockets;James;Arthritis Rheum,2010
2. Specific interaction between genotype, smoking and autoimmunity to citrullinated [alpha]-enolase in the etiology of rheumatoid arthritis;Mahdi;Nat Genet,2009
3. Gene–environment interaction influences the reactivity of autoantibodies to citrullinated antigens in rheumatoid arthritis;Van der Woude;Nat Genet,2010
4. Citrullinated vimentin as an important antigen in immune complexes from synovial fluid of rheumatoid arthritis patients with antibodies against citrullinated proteins;Van Steendam;Arthritis Res Ther,2010
5. Antibodies to several citrullinated antigens are enriched in the joints of rheumatoid arthritis patients;Snir;Arthritis Rheum,2010
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