Expression of Smad Protein by Normal Skin Fibroblasts and Hypertrophic Scar Fibroblasts in Response to Transforming Growth Factor β1-Reference-Cited by-同舟云学术

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Expression of Smad Protein by Normal Skin Fibroblasts and Hypertrophic Scar Fibroblasts in Response to Transforming Growth Factor β1

Published:2008-09 Issue:9 Volume:34 Page:1216-1225
ISSN:1076-0512
Container-title:Dermatologic Surgery
language:en
Short-container-title:Dermatologic Surgery

Author:

XIE JU-LIN,QI SHAO-HAI,PAN SHU,XU YING-BIN,LI TIAN-ZENG,LIU XU-SHENG,LIU PO

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Dermatology,General Medicine,Surgery

Reference36 articles.

1. Mechanisms of tissue repair: from wound healing to fibrosis

2. Keloids and hypertrophic scars: Review and treatment strategies

3. Increased Types I and III Collagen and Transforming Growth Factor-β1 mRNA and Protein in Hypertrophic Burn Scar

4. Biochemical Composition of the Connective Tissue in Keloids and Analysis of Collagen Metabolism in Keloid Fibroblast Cultures

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1. Human Keratinocyte-Derived Exosomal MALAT1 Promotes Diabetic Wound Healing by Upregulating MFGE8 via microRNA-1914-3p;International Journal of Nanomedicine;2023-02

2. Circular RNA HECTD1 knockdown inhibits transforming growth factor-beta/ small mothers against decapentaplegic (TGF-β/Smad) signaling to reduce hypertrophic scar fibrosis;Bioengineered;2022-03-01

3. Smad7 down-regulation via ubiquitin degradation mediated by Smurf2 in fibroblasts of hypertrophic scars in burned patients;Burns;2021-09

4. A Review of Hypertrophic Scar and Keloid Treatment and Prevention in the Pediatric Population: Where Are We Now?;Advances in Wound Care;2021-07-14

5. Burns;Braddom's Physical Medicine and Rehabilitation;2021

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