Induction of G1 arrest in glioma cells by T11TS is associated with upregulation of Cip1/Kip1 and concurrent downregulation of cyclin D (1 and 3)-Reference-Cited by-同舟云学术

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Induction of G1 arrest in glioma cells by T11TS is associated with upregulation of Cip1/Kip1 and concurrent downregulation of cyclin D (1 and 3)

Published:2010-01 Issue:1 Volume:21 Page:53-64
ISSN:0959-4973
Container-title:Anti-Cancer Drugs
language:en
Short-container-title:

Author:

Acharya Sagar,Chatterjee Sirshendu,Kumar Pankaj,Bhattacharjee Malabika,Chaudhuri Suhnrita,Chaudhuri Swapna

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cancer Research,Pharmacology (medical),Pharmacology,Oncology

Reference51 articles.

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2. Cyclin-dependent kinases: engines, clocks, and microprocessors;Morgan;Annu Rev Cell Dev Biol,1997

3. Targets of oncogenes and tumor suppressors: key for understanding basic mechanisms of carcinogenesis;Kopnin;Biochemistry,2000

4. Cyclin D1 is dispensable for G1 control in retinoblastoma gene-deficient cells independently of CDK4 activity;Lukas;Mol Cell Biol,1995

5. Retinoblastoma-protein-dependent cell-cycle inhibition by the tumor suppressor p16;Lukas;Nature,1995

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5. T11TS immunotherapy repairs PI3K‐AKT signaling in T‐cells: Clues toward enhanced T‐cell survival in rat glioma model;Journal of Cellular Physiology;2017-07-11

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