Protein misfolding, congophilia, oligomerization, and defective amyloid processing in preeclampsia-Reference-Cited by-同舟云学术

Protein misfolding, congophilia, oligomerization, and defective amyloid processing in preeclampsia

Published:2014-07-16 Issue:245 Volume:6 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Buhimschi Irina A.¹²,Nayeri Unzila A.²,Zhao Guomao¹,Shook Lydia L.²,Pensalfini Anna³,Funai Edmund F.⁴,Bernstein Ira M.⁵,Glabe Charles G.⁶⁷,Buhimschi Catalin S.²⁴

Affiliation:

1. Center for Perinatal Research, The Research Institute at Nationwide Children’s Hospital and Department of Pediatrics, The Ohio State University College of Medicine, Columbus, OH 43215, USA.

2. Department of Obstetrics, Gynecology and Reproductive Sciences, Yale University School of Medicine, New Haven, CT 06520, USA.

3. Center for Dementia Research, Nathan Kline Institute for Psychiatric Research and Department of Psychiatry, New York University School of Medicine, New York, NY 10016, USA.

4. Department of Obstetrics and Gynecology, The Ohio State University College of Medicine, Columbus, OH 43210, USA.

5. Department of Obstetrics, Gynecology and Reproductive Sciences, University of Vermont College of Medicine, Burlington, VT 05405, USA.

6. Department of Molecular Biology and Biochemistry, University of California, Irvine, Irvine, CA 92617, USA.

7. Department of Biochemistry and Experimental Biochemistry Unit, King Abdulaziz University, Jeddah 22254, Saudi Arabia.

Abstract

Preeclampsia is characterized by protein misfolding, aggregation, and amyloid-like plaque formation, suggesting that this pregnancy-related condition shares pathophysiologic features with protein conformational disorders.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference70 articles.

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4. Pre-eclampsia

5. Preeclampsia, Hypoxia, Thrombosis, and Inflammation

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