IgH sequences in common variable immune deficiency reveal altered B cell development and selection-Reference-Cited by-同舟云学术

IgH sequences in common variable immune deficiency reveal altered B cell development and selection

Published:2015-08-26 Issue:302 Volume:7 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Roskin Krishna M.¹,Simchoni Noa²,Liu Yi¹³,Lee Ji-Yeun¹,Seo Katie¹,Hoh Ramona A.¹,Pham Tho¹,Park Joon H.²,Furman David⁴⁵,Dekker Cornelia L.⁶,Davis Mark M.⁴⁵⁷,James Judith A.⁸,Nadeau Kari C.⁶,Cunningham-Rundles Charlotte²,Boyd Scott D.¹

Affiliation:

1. Department of Pathology, Stanford University, Stanford, CA 94305, USA.

2. Departments of Medicine and Pediatrics, Icahn School of Medicine at Mount Sinai, New York, NY 10029, USA.

3. Biomedical Informatics Training Program, Stanford University, Stanford, CA 94305, USA.

4. Department of Microbiology and Immunology, Stanford University, Stanford, CA 94305, USA.

5. Institute for Immunity, Transplantation and Infection, Stanford University, Stanford, CA 94305, USA.

6. Departments of Medicine, Pediatrics, and Otolaryngology, Stanford University, Stanford, CA 94305, USA.

7. Howard Hughes Medical Institute, Stanford University, Stanford, CA 94305, USA.

8. Arthritis and Clinical Immunology Research Program, Oklahoma Medical Research Foundation and Oklahoma Clinical and Translational Science Institute and Department of Medicine, University of Oklahoma Health Sciences Center, Oklahoma City, OK 73104, USA.

Abstract

Deep sequence analysis of the IgH repertoires of common variable immune deficiency patients highlights phenotypic features of the disorder and potential disease mechanisms.

Funder

NIH

Ellison Medical Foundation

Jeffrey Modell Foundation

Stanford Center for Clinical and Translational Education and Research NIH

David S. Gottesman Immunology Chair

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

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5. Common variable immunodeficiency disorders: division into distinct clinical phenotypes

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