NAAA-regulated lipid signaling governs the transition from acute to chronic pain-Reference-Cited by-同舟云学术

NAAA-regulated lipid signaling governs the transition from acute to chronic pain

Published:2021-10-22 Issue:43 Volume:7 Page:
ISSN:2375-2548
Container-title:Science Advances
language:en
Short-container-title:Sci. Adv.

Author:

Fotio Yannick¹^ORCID,Jung Kwang-Mook¹^ORCID,Palese Francesca¹^ORCID,Obenaus Andre²^ORCID,Tagne Alex Mabou¹^ORCID,Lin Lin¹^ORCID,Rashid Tarif Ibne¹^ORCID,Pacheco Romario³^ORCID,Jullienne Amandine²^ORCID,Ramirez Jade¹^ORCID,Mor Marco⁴^ORCID,Spadoni Gilberto⁵^ORCID,Jang Cholsoon⁶^ORCID,Hohmann Andrea G.³^ORCID,Piomelli Daniele¹^ORCID

Affiliation:

1. Department of Anatomy and Neurobiology, University of California Irvine, Irvine, CA 92697, USA.

2. Department of Pediatrics, University of California Irvine, Irvine, CA 92697, USA.

3. Psychological and Brain Sciences, Program in Neuroscience, and Gill Center for Biomolecular Science, Indiana University, Bloomington, IN 47401, USA.

4. Dipartimento di Scienze degli Alimenti e del Farmaco, Università di Parma, 43124 Parma, Italy.

5. Dipartimento di Scienze Biomolecolari, Università di Urbino “Carlo Bo,” 61029 Urbino, Italy.

6. Department of Biological Chemistry, University of California Irvine, Irvine, CA 92697, USA.

Abstract

The enzyme NAAA is a critical control node that offers a druggable target to prevent the transition from acute to chronic pain.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference87 articles.

1. The Transition of Acute Postoperative Pain to Chronic Pain: An Integrative Overview of Research on Mechanisms

2. Pain vulnerability: a neurobiological perspective

3. Cellular and Molecular Mechanisms of Pain

4. Pain regulation by non-neuronal cells and inflammation

5. Transition to chronic pain: opportunities for novel therapeutics

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