CTNNB1/ β -catenin dysfunction contributes to adiposity by regulating the cross-talk of mature adipocytes and preadipocytes

Author:

Chen Maopei1ORCID,Lu Peng12ORCID,Ma Qinyun1ORCID,Cao Yanan1ORCID,Chen Na1,Li Wen1,Zhao Shaoqian1,Chen Banru1,Shi Juan1,Sun Yingkai1,Shen Hongbin3,Sun Liangdan4,Shen Juan5,Liao Qijun5,Zhang Yifei1,Hong Jie1,Gu Weiqiong1,Liu Ruixin1ORCID,Ning Guang12ORCID,Wang Weiqing1ORCID,Wang Jiqiu1ORCID

Affiliation:

1. Shanghai National Clinical Research Center for Metabolic Diseases, Key Laboratory for Endocrine and Metabolic Diseases of Chinese Health Commission, Department of Endocrinology and Metabolism, Shanghai Key Laboratory for Endocrine Tumors, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine (SJTUSM), Shanghai, China.

2. CAS Key Laboratory of Tissue Microenvironment and Tumor, Shanghai Institute of Nutrition and Health, Shanghai Institutes for Biological Sciences, University of Chinese Academy of Sciences, Chinese Academy of Sciences (CAS), Shanghai, China.

3. Institute of Image Processing and Pattern Recognition, SJTU, Shanghai, China.

4. Institute of Dermatology and Department of Dermatology, No.1 Hospital, Anhui Medical University, Hefei, China.

5. BGI Genomics, BGI-Shenzhen, Shenzhen, China.

Abstract

β-catenin regulates the cross-talk among mature adipocytes, macrophages, and preadipocytes under overnutrition.

Funder

National Natural Science Foundation of China

Foundation of Shanghai Municipal Commission of Health and Family Planning

National Key Research and Development Program of China

Shanghai Rising-Star Program

Shanghai Municipal Education Commission-Gaofeng Clinical Medicine Grant Support

973 Foundation

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

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