Interferon-driven deletion of antiviral B cells at the onset of chronic infection-Reference-Cited by-同舟云学术

Interferon-driven deletion of antiviral B cells at the onset of chronic infection

Published:2016-10-21 Issue:4 Volume:1 Page:
ISSN:2470-9468
Container-title:Science Immunology
language:en
Short-container-title:Sci. Immunol.

Author:

Fallet Benedict¹,Narr Kerstin¹,Ertuna Yusuf I.¹,Remy Melissa¹,Sommerstein Rami²,Cornille Karen¹,Kreutzfeldt Mario²³,Page Nicolas²,Zimmer Gert⁴,Geier Florian⁵,Straub Tobias⁶,Pircher Hanspeter⁶,Larimore Kevin⁷⁸,Greenberg Philip D.⁷⁸,Merkler Doron²³,Pinschewer Daniel D.¹

Affiliation:

1. Division of Experimental Virology, Department of Biomedicine, University of Basel, 4003 Basel, Switzerland.

2. Department of Pathology and Immunology, Geneva Faculty of Medicine, Geneva University Hospital, 1211 Geneva 4, Switzerland.

3. Division of Clinical Pathology, Geneva University Hospital, 1 rue Michel Servet, 1211 Geneva 4, Switzerland.

4. Institute of Virology and Immunology, 3147 Mittelhäusern, Switzerland.

5. Department of Biomedicine, Bioinformatics Core Facility, University Hospital Basel, 4031 Basel, Switzerland.

6. Department for Medical Microbiology and Hygiene, Institute for Immunology, University Medical Center Freiburg, 79104 Freiburg, Germany.

7. Fred Hutchinson Cancer Research Center, University of Washington, Seattle, WA 98109, USA.

8. Department of Immunology, University of Washington, Seattle, WA 98109, USA.

Abstract

Interferon-driven inflammation in chronic viral infection blocks generation of sustained B cell responses. See related Research Articles by Moseman et al. and Sammicheli et al.

and a Focus by Laidlaw et al.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine,Immunology

Reference68 articles.

1. B-cell exhaustion in HIV infection

2. Enhanced B-cell differentiation and reduced proliferative capacity in chronic hepatitis C and chronic hepatitis B virus infections

3. Atypical Memory B Cells Are Greatly Expanded in Individuals Living in a Malaria-Endemic Area

4. Alterations in Peripheral Blood B Cell Subsets and Dynamics of B Cell Responses during Human Schistosomiasis

5. Patients with Tuberculosis Have a Dysfunctional Circulating B-Cell Compartment, Which Normalizes following Successful Treatment

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