Cellular context of IL-33 expression dictates impact on anti-helminth immunity

Author:

Hung Li-Yin1ORCID,Tanaka Yukinori2ORCID,Herbine Karl1ORCID,Pastore Christopher1ORCID,Singh Brenal3ORCID,Ferguson Annabel1,Vora Nisha1,Douglas Bonnie1ORCID,Zullo Kelly1,Behrens Edward M.4,Li Hui Tan Tiffany5ORCID,Kohanski Michael A.5ORCID,Bryce Paul6ORCID,Lin Cailu7ORCID,Kambayashi Taku3,Reed Danielle R.7ORCID,Brown Breann L.8ORCID,Cohen Noam A.579,Herbert De’Broski R.1ORCID

Affiliation:

1. Department of Pathobiology, School of Veterinary Medicine, University of Pennsylvania, Philadelphia, PA 19104, USA.

2. Department of Dental Anesthesiology and Pain Management, Tohoku University Hospital, Sendai, Miyagi 980-8574, Japan.

3. Department of Pathology and Laboratory Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA 19104, USA.

4. Division of Rheumatology, Children’s Hospital of Philadelphia, Philadelphia, PA 19104, USA.

5. Department of Otorhinolaryngology–Head and Neck Surgery, Perelman School of Medicine at The University of Pennsylvania, Philadelphia, PA 19104, USA.

6. Immunology and Inflammation Therapeutic Area, Sanofi US, Cambridge, MA 02319, USA.

7. Monell Chemical Senses Center, Philadelphia, PA 19104, USA.

8. Department of Biochemistry, Center for Structural Biology, Vanderbilt University, Nashville, TN 37232, USA.

9. Michael J. Crescenz Veterans Affairs Medical Center Surgical Service, Philadelphia, PA 19104, USA.

Abstract

The pore-forming protein perforin-2 facilitates IL-33 release from dendritic cells.

Funder

National Institutes of Health

Burroughs Wellcome Fund

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine,Immunology

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